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Learn MoreTo study whether acute inhibition of DOT1L induces global chromatin states alterations, we profile and compare the transcriptome, chromatin accessibility and epigenome (H3K4me3, H3K4me1, H3K27ac, H3K27me3, H3K36me3, H3K9me3, H3K79me2) of mESC and ES-derived NPC, treated with DMSO or EPZ5676. SOURCE: Thomas Manke (manke@ie-freiburg.mpg.de) - Max Planck Institute of Immunobiology and Epigenetics
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